There is a perception of conflicting data and opinions among research laboratories that are studying periodontal microbiology. Subgingival microflora and periodontal disease. Others studies sought differences in the microorganisms in subgingival plaque samples taken from subjects with clinically different forms of periodontal disease or periodontal health. 1880–1920), when the etiologic agents of many medically important infections were determined. 32. Slideshare uses cookies to improve functionality and performance, and to provide you with relevant advertising. 36. Microbiology of periodontal disease . The search started in the “golden age of microbiology” (approx. It is found in the oral cavity, where it is implicated in periodontal disease, as well as in the upper gastrointestinal tract, the respiratory tract and the colon.It has been isolated from women with bacterial vaginosis. Microbial interactionswith the host in periodontal diseases 7. Diabetes mellitus ppt p. gingivalis: Hygienist salary chlorhexidine rinse: hydrogen peroxide mouthwash antioxidants ppt: Metronidazole dosage implant center greenville: Peptostreptococcus micros erectile dysfunction: Microbiology decay aetiology progression: amoxicillin dosage porphyromonas gingivalis: Chlorhexidine rinse myocardial infarction A, Streptococcus mitis are gram-positive, fast-growing, facultatively anaerobic bacteria that are easy to culture on a blood-agar plate. LVH and WEC Moore [14] . Dentoalveolar infections . Microbiology of. Corn Cob Formation (Listgarten et al, 1973)– Central Gram Negative Filamentous Core Supports Outer Coccal Cells Which Are Firmly Attached By Inter-bacterial Adherence Or Coaggregation. Periodontal disease; ; Delivery Systems. Many investigators claimed success in controlling advanced destructive periodontal disease by combining local and systemic therapy. Within a short period of time, signs or symptoms of a disease appear at the site of introduction or elsewhere in the individual. Detecting pathologies at their earliest stages can significantly affect patient discomfort, prognosis, therapeutic intervention, survival rates, and recurrence. Lancefield groups : A to V without I and T. In acute infections, an agent is acquired by exposure to an individual harbouring that agent or from the environment. Periodontal microbiology 1. MicroBioLogy of Periodontal Diseases - Free download as Powerpoint Presentation (.ppt), PDF File (.pdf), Text File (.txt) or view presentation slides online. ... PPT Chapter 13 Asthma 35 Terms. In certain ways, periodontal diseases may be among the most unusual infections of the human.In contrast to the outer surface of most parts of the body, the outer layers of the tooth do not “shed” and thus microbial colonization (accumulation) is facilitated. Human fetus is sterile but after passing through birth canal it acquires vaginal and fecal microorganisms By 2nd day Anaerobic flora can be detected in infant edentulous mouth Dr Saif Khan 209/10/13 3. Oral microbiology ppt دخول. We use your LinkedIn profile and activity data to personalize ads and to show you more relevant ads. Looks like you’ve clipped this slide to already. APIdays Paris 2019 - Innovation @ scale, APIs as Digital Factories' New Machi... No public clipboards found for this slide. Clarus Dental Periodontal Disease Treatment Center - Periodontal disease is infection of the gums and if not treated on time can contribute to receding gums. Periodontal diseases are infections that are caused by microorganisms that colonize the tooth surface at or below the gingival margin. If you continue browsing the site, you agree to the use of cookies on this website. See our User Agreement and Privacy Policy. If you continue browsing the site, you agree to the use of cookies on this website. Microbiology of periodontal disease- part 2, Customer Code: Creating a Company Customers Love, Be A Great Product Leader (Amplify, Oct 2019), Trillion Dollar Coach Book (Bill Campbell). 4.7.4 Proteases and DNases. Thus, daily experience suggests that colonization by a pathogen is rapidly followed by expression of disease. During the late 1950s, a group of clinicians, sometimes referred to as “plaque evangelists”, heavily emphasized the need for plaque control in the prevention and treatment of periodontal diseases.Thus, once again bacteria were thought to play a role in the etiology of destructive periodontal disease, but as non-specific causative agents. Now customize the name of a clipboard to store your clips. It may form on teeth, mucosa or other solid surfaces. Procedures such as dental scaling can radically alter the composition of the periodontal pocket flora of diseased sites and shift the balance in favour of colonization of such sites by flora that are associated with health. Individual Micro colonies can consist of single species but are frequently composed of several different species. In essence, this is a natural cleansing mechanism. 33. Periodontal pathogens such as P. gingivalis, Tannerella forsythia, and Treponema denticola are asaccharolytic and strongly proteolytic. These deposits can be readily visualised with vegetable or synthetic dyes in disclosing solutions, and can become calcified to form calculus. Some studies attempted to determine whether specific microorganisms were found in lesion sites as compared to healthy sites. Figure 8-1 Various periodontal and cariogenic species grown on agar plates. MICROBIOLOGY OF PERIODONTAL DISEASE ... Plaque control Root surface debridement Periodontal surgery Prudent use of antimicrobial agents Page ... – A free PowerPoint PPT presentation (displayed as a Flash slide show) on PowerShow.com - id: 45b7e3-YTQ1N J Bacteriol 2010;192:5002-17. Twenty-four patients (10 males and 14 females, aged 14.6 ± 1.0 years) were included in this investigation. 37. Slideshare uses cookies to improve functionality and performance, and to provide you with relevant advertising. Slideshare uses cookies to improve functionality and performance, and to provide you with relevant advertising. (Gram-negative bacteria,short, pleomorphic rods) Fusobacterium sp. Socransky SS, Haffajee AD, Cugini MA, Smith C, Kent RL Jr. Microbial complexes in subgingival plaque. Principles of infection control . Pathology of periodontal disease A. Hasan1 and R. M. Palmer*2 rinsed away. In response to this perception I was asked to discuss recent literature in this field at the 1986 Annual Meeting of the American Association of Dental Research. Start studying Perio Microbiology Powerpoint 2. This means that this microbiota lives in harmony with its host, but under specific conditions (increased mass and/or pathogenicity, suppression of commensal or beneficial bacteria and/or reduced host response), disease can occur. If you continue browsing the site, you agree to the use of cookies on this website. Dr. Fletcher is currently the Assistant Dean for Graduate Student Affairs in the School of Medicine at Loma Linda University. From an ecologic viewpoint, the oral cavity, which communicates with the pharynx, should be considered as an “open growth system” with an uninterrupted ingestion and removal of microorganisms and their nutrients. The human oral microbiome. Microbiology of Periodontal Diseases. 1979 Oct;6(5):351-82. doi: 10.1111/j.1600-051x.1979.tb01935.x. One of the most important mechanisms is shedding. Bacteroides sp. The proposal of the “ecological plaque hypothesis” in 2003 was a significant development in the conceptual understanding of the subgingival environment and how the local environment can influence bacterial composition. You can change your ad preferences anytime. Microbial Etiology of Periodontal Disease, Dr. Lee 2 Dental plaque biofilm infection Ecological point of view Ecological community evolved for survival as a whole Complex community of more than 400 bacterial species Dynamic equilibrium between bacteria and a host defense Adopted survival strategies favoring growth in plaque “Selection” of “pathogenic” bacteria among microbial community Pathogenesis of paque associated periodontal diseases 8. 34. The search for the etiologic agents of periodontal diseases has been in progress for over a century. The soft tissue surfaces employ a variety of mechanisms to prevent adhesion of pathogenic organisms. 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